The autophagy-activating kinase ULK1 mediates clearance of free α-globin in β-thalassemia
Christophe Lechauve(St. Jude Children's Research Hospital), Julia Keith(St. Jude Children's Research Hospital), Eugene Khandros(Children's Hospital of Philadelphia), Stephanie Fowler(St. Jude Children's Research Hospital), Kalin Mayberry(St. Jude Children's Research Hospital), Abdullah Freiwan(St. Jude Children's Research Hospital), Christopher S. Thom(Children's Hospital of Philadelphia), Paola Delbini(University of Milan), Emilio Boada-Romero(St. Jude Children's Research Hospital), Jingjing Zhang(St. Jude Children's Research Hospital), Irene Motta(University of Milan), Heather Tillman(St. Jude Children's Research Hospital), Maria Domenica Cappellini(University of Milan), Mondira Kundu(St. Jude Children's Research Hospital), Mitchell J. Weiss(St. Jude Children's Research Hospital)
Cited by 62Open Access
Abstract
cells of β-thalassemic individuals. Our findings define a drug-regulatable pathway for ameliorating β-thalassemia.
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