DRP-1 functions independently of mitochondrial structural perturbations to facilitate BH3 mimetic-mediated apoptosis
Mateus Milani(University of Liverpool), Shankar Varadarajan(University of Liverpool)
Cited by 33
Related Papers
BH3-only proteins are dispensable for apoptosis induced by pharmacological inhibition of both MCL-1 and BCL-XL
|Cell Death and Differentiation|2018|68
Maritoclax and dinaciclib inhibit MCL-1 activity and induce apoptosis in both a MCL-1-dependent and -independent manner
|Oncotarget|2015|46
The transrepression arm of glucocorticoid receptor signaling is protective in mutant huntingtin-mediated neurodegeneration
|Cell Death and Differentiation|2015|34
Exploring the potential of BH3 mimetic therapy in squamous cell carcinoma of the head and neck
|Cell Death and Disease|2019|29
Novel roles of RTN4 and CLIMP-63 in regulating mitochondrial structure, bioenergetics and apoptosis
|Cell Death and Disease|2022|29