Lipopolysaccharide endotoxemia induces amyloid-β and p-tau formation in the rat brain.
Liming Wang(Fujian Agriculture and Forestry University), Kathryn A. Morton, Jeffrey T. Yap(Institute for Tourism), John M. Hoffman(University of California, Los Angeles), Rosana Souza Rodrigues(Universidade Federal do Rio de Janeiro), Ryan A. Kirk(University of Utah), Rheal A. Towner(Oklahoma Medical Research Foundation), Fernando A. Bozza(D’Or Institute for Research and Education), Joshua A. Sonnen(Rush University), Qi Wu(University of Utah), Ahmed Ebada Salem(University of Utah), Kevin P. Horn(University of Washington)
PubMed
January 1, 2018
Cited by 65
Related Papers
Correlation of Alzheimer Disease Neuropathologic Changes With Cognitive Status: A Review of the Literature
|Journal of Neuropathology & Experimental Neurology|2012|2.1k
A minimal common outcome measure set for COVID-19 clinical research
|The Lancet Infectious Diseases|2020|1.8k
Positron Emission Tomography in Evaluation of Dementia
|JAMA|2001|952
Allele-defined genome of the autopolyploid sugarcane Saccharum spontaneum L.
|Nature Genetics|2018|789