Contribution of DNMT1 to Neuropathic Pain Genesis Partially through Epigenetically Repressing <i>Kcna2</i> in Primary Afferent Neurons
Yuan‐Xiang Tao(Rutgers, The State University of New Jersey)
Cited by 105
Related Papers
DNA methyltransferase DNMT3a contributes to neuropathic pain by repressing Kcna2 in primary afferent neurons
|Nature Communications|2017|233
DNMT3a‐triggered downregulation of K<sub>2p</sub>1.1 gene in primary sensory neurons contributes to paclitaxel‐induced neuropathic pain
|International Journal of Cancer|2019|104
Protein Kinase B/Akt Is Required for Complete Freund's Adjuvant-Induced Upregulation of Nav1.7 and Nav1.8 in Primary Sensory Neurons
|Journal of Pain|2013|59
MBD1 Contributes to the Genesis of Acute Pain and Neuropathic Pain by Epigenetic Silencing of<i>Oprm1</i>and<i>Kcna2</i>Genes in Primary Sensory Neurons
|Journal of Neuroscience|2018|57
Role of MicroRNA-143 in Nerve Injury-Induced Upregulation of Dnmt3a Expression in Primary Sensory Neurons
|Frontiers in Molecular Neuroscience|2017|40