Preso regulates NMDA receptor-mediated excitotoxicity via modulating nitric oxide and calcium responses after traumatic brain injuryPeng Luo(Xijing Hospital), Hongbing Lu(Air Force Medical University)Cell Death and DiseaseJune 24, 201910.1038/s41419-019-1731-xCited by 42SaveCiteExport RISWatch citationsRelated PapersHomer1 knockdown protects dopamine neurons through regulating calcium homeostasis in an in vitro model of Parkinson's disease|Cellular Signalling|2013|66Preso enhances mGluR1-mediated excitotoxicity by modulating the phosphorylation of mGluR1-Homer1 complex and facilitating an ER stress after traumatic brain injury|Cell Death Discovery|2024|3