Determination of PD-L1 Expression in Circulating Tumor Cells of NSCLC Patients and Correlation with Response to PD-1/PD-L1 Inhibitors

Melanie Janning(Universität Hamburg), Franca Kobus(Universität Hamburg), Anna Babayan(Universität Hamburg), Harriet Wikman(Universität Hamburg), Janna-Lisa Velthaus(Universität Hamburg), Sonja Bergmann(Universität Hamburg), Stefanie Schatz(Institut für Hämatopathologie Hamburg), Markus Falk(Institut für Hämatopathologie Hamburg), Lars-Arne Berger(Universität Hamburg), Lisa-Marie Böttcher(Universität Hamburg), Sarina Päsler(Universität Hamburg), Tobias M. Gorges(Universität Hamburg), Linda O′Flaherty(Boehringer Ingelheim (Germany)), Claudia Hille(Universität Hamburg), Simon A. Joosse(Universität Hamburg), Ronald Simon(Universität Hamburg), Markus Tiemann(Institut für Hämatopathologie Hamburg), Carsten Bokemeyer(Universität Hamburg), Martin Reck(LungenClinic Grosshansdorf), Sabine Riethdorf(Universität Hamburg), Klaus Pantel(Universität Hamburg), Sonja Loges(Universität Hamburg)
Cancers
June 17, 2019
Cited by 135Open Access
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Abstract

Circulating tumor cells (CTCs) hold great potential to answer key questions of how non-small cell lung cancer (NSCLC) evolves and develops resistance upon anti-PD-1/PD-L1 treatment. Currently, their clinical utility in NSCLC is compromised by a low detection rate with the established, Food and Drug Administration (FDA)-approved, EpCAM-based CellSearch® System. We tested an epitope-independent method (ParsortixTM system) and utilized it to assess PD-L1 expression of CTCs from NSCLC patients. We prospectively collected 127 samples, 97 of which were analyzed with the epitope-independent system in comparison to the CellSearch system. CTCs were determined by immunocytochemistry as intact, nucleated, CD45−, pankeratins (K)+ cells. PD-L1 status of CTCs was evaluated from 89 samples. With the epitope-independent system, ≥1 CTC per blood sample was detected in 59 samples (61%) compared to 31 samples (32%) with the EpCAM-based system. Upon PD-L1 staining, 47% of patients harbored only PD-L1+CTCs, 47% had PD-L1+ and PD-L1−CTCs, and only 7% displayed exclusively PD-L1−CTCs. The percentage of PD-L1+CTCs did not correlate with the percentage of PD-L1+ in biopsies determined by immunohistochemistry (p = 0.179). Upon disease progression, all patients showed an increase in PD-L1+CTCs, while no change or a decrease in PD-L1+CTCs was observed in responding patients (n = 11; p = 0.001). Our data show a considerable heterogeneity in the PD-L1 status of CTCs from NSCLC patients. An increase of PD-L1+CTCs holds potential to predict resistance to PD-1/PD-L1 inhibitors.


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