Integrative Analysis Identifies Four Molecular and Clinical Subsets in Uveal Melanoma

Abstract

(Cancer Cell 32, 204–220; August 14, 2017) During the course of revising the manuscript, the authors inadvertently did not include the reference to a paper by Royer-Bertrand et al. in the reference list as well as the corresponding citation in the discussion of copy number variants in uveal melanoma and their consistency in high-risk primary and metastatic tumors. Royer-Bertrand et al. should have been cited in the second paragraph under the Discussion section as follows: “Prior studies have shown poorer clinical outcomes to be associated with higher chromosome 8q copy number (Royer-Bertrand et al., 2016Royer-Bertrand B. Torsello M. Rimoldi D. El Zaoui I. Cisarova K. Pescini-Gobert R. Raynaud F. Zografos L. Schalenbourg A. Speiser D. et al.Comprehensive genetic landscape of uveal melanoma by whole-genome sequencing.Am. J. Hum. Genet. 2016; 99: 1190-1198Abstract Full Text Full Text PDF PubMed Scopus (107) Google Scholar; Caines et al., 2015; Cassoux et al., 2014; Versluis et al., 2015).” The reference is now included below and has been added to the online version of the article, along with the citation. The authors apologize for the omission and any confusion it may have caused. Integrative Analysis Identifies Four Molecular and Clinical Subsets in Uveal MelanomaRobertson et al.Cancer CellAugust 14, 2017In BriefRobertson et al. analyze 80 uveal melanomas (UM) and divide poor-prognosis monosomy 3 UM into subsets with divergent genomic aberrations, transcriptional features, and clinical outcomes. Somatic copy number changes and DNA methylation profiles separate better-prognosis disomy 3 UM into low or intermediate risk. Full-Text PDF Open Access