Defects in Efflux ( oprM ), β-Lactamase ( ampC ), and Lipopolysaccharide Transport ( lptE ) Genes Mediate Antibiotic Hypersusceptibility of Pseudomonas aeruginosa Strain Z61

Xiaoyu Shen(Novartis (United States)), Charles R. Dean(University of Virginia Health System), S. Whitney Barnes(Genomics Institute of the Novartis Research Foundation), Nicole V. Johnson(The University of Texas at Austin), Naomi N. K. Kreamer(California Institute of Technology), David A. Six(Duke Medical Center), John R. Walker(Genomics Institute of the Novartis Research Foundation), Angela Woods(Moderna Therapeutics (United States))

Antimicrobial Agents and Chemotherapy

April 24, 2019

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Defects in Efflux ( <i>oprM</i> ), β-Lactamase ( <i>ampC</i> ), and Lipopolysaccharide Transport ( <i>lptE</i> ) Genes Mediate Antibiotic Hypersusceptibility of <i>Pseudomonas aeruginosa</i> Strain Z61