Genome-wide analysis identifies NR4A1 as a key mediator of T cell dysfunction
Xindong Liu(Chinese Academy of Sciences), Yun Wang(Army Medical University), Huiping Lu(Tsinghua University), Jing Li(Tsinghua University), Xiaowei Yan(Institute for Systems Biology), Minglu Xiao(Army Medical University), Jing Hao(Tsinghua University), Andrei Alekseev(The University of Texas MD Anderson Cancer Center), Hiep Khong(The University of Texas MD Anderson Cancer Center), Tenghui Chen(The University of Texas MD Anderson Cancer Center), Rui Huang(Army Medical University), Jin Wu(Southwest Hospital), Qiwen Zhao(Army Medical University), Qi Wu(Tsinghua University), Senlin Xu(Southwest Hospital), Xiaohu Wang(Tsinghua University), Wei Jin(Tsinghua University), Shi‐Cang Yu(Southwest Hospital), Yan Wang(Southwest Hospital), Lai Wei(Sun Yat-sen University), Aibo Wang(The University of Texas MD Anderson Cancer Center), Bo Zhong(Wuhan University), Ling Ni(Tsinghua University), Xiaolong Liu(Chinese Academy of Sciences), Roza Nurieva(The University of Texas MD Anderson Cancer Center), Lilin Ye(Army Medical University), Qiang Tian(Institute for Systems Biology), Xiu‐Wu Bian(Army Medical University), Chen Dong(Tsinghua University)
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