Transcriptional repressor REST drives lineage stage–specific chromatin compaction at <i>Ptch1</i> and increases AKT activation in a mouse model of medulloblastoma
Tara Dobson(The University of Texas MD Anderson Cancer Center), Rong-Hua Tao(The University of Texas MD Anderson Cancer Center), Jyothishmathi Swaminathan(The University of Texas MD Anderson Cancer Center), Shinji Maegawa(The University of Texas MD Anderson Cancer Center), Shavali Shaik(The University of Texas MD Anderson Cancer Center), Javiera Bravo‐Alegria(The University of Texas MD Anderson Cancer Center), Ajay Sharma(The University of Texas MD Anderson Cancer Center), Bridget Kennis(The University of Texas MD Anderson Cancer Center), Yanwen Yang(The University of Texas MD Anderson Cancer Center), Keri Callegari(The University of Texas MD Anderson Cancer Center), Amanda R. Haltom(The University of Texas MD Anderson Cancer Center), Pete Taylor(The University of Texas MD Anderson Cancer Center), Mari Kogiso(Baylor College of Medicine), Lin Qi(Baylor College of Medicine), Soumen Khatua(The University of Texas MD Anderson Cancer Center), Stewart Goldman(Northwestern University), Rishi Lulla(Northwestern University), Jason Fangusaro(Northwestern University), Tobey J. MacDonald(Emory University), Xiao‐Nan Li(Northwestern University), Cynthia Hawkins(Hospital for Sick Children), Veena Rajaram(Southwestern Medical Center), Vidya Gopalakrishnan(The University of Texas MD Anderson Cancer Center)
Cited by 32Open Access
Abstract
silencing, and AKT activation in MB tissues reveal potential subgroup-specific therapeutic targets for MB patients.
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