Aging-like Spontaneous Epigenetic Silencing Facilitates Wnt Activation, Stemness, and BrafV600E-Induced Tumorigenesis
Yong Tao(Shanghai Ninth People's Hospital), Hariharan Easwaran(Johns Hopkins University), Nicholas C. Zachos(Johns Hopkins University), Stephen M. Brown(Johns Hopkins University), Himani Vaidya(Temple University), Julie In(University of New Mexico), Izaskun Mallona(Institut d'Investigació en Ciències de la Salut Germans Trias i Pujol), Miguel A. Peinado(Badalona Serveis Assistencials), Elana J. Fertig(Bloomberg (United States)), Shinji Maegawa(The University of Texas MD Anderson Cancer Center), Jai Thakor(Center for Special Minimally Invasive and Robotic Surgery), Yuezheng Zhang(Chinese Academy of Sciences), Zhihao Lü(Nanjing Tech University), Wenbing Xie(Johns Hopkins University), Daniel Petkovich(Johns Hopkins University), Cynthia A. Zahnow(Johns Hopkins University), Stephen B. Baylin(Johns Hopkins University), Jean‐Pierre J. Issa(Coriell Institute For Medical Research), Genevieve Stein-O’Brien(Johns Hopkins University), Yi Cai(Johns Hopkins University), Xiangqian Kong(University of Science and Technology of China), Byunghak Kang(Johns Hopkins University), Ray-Whay Chiu Yen(Johns Hopkins University), Xiaojian Shao(University of Toronto), Nita Ahuja(Johns Hopkins University), Yuba R. Bhandari(Frederick National Laboratory for Cancer Research)
Cited by 191
Related Papers
Incidence and functional consequences of <i>hMLH1</i> promoter hypermethylation in colorectal carcinoma
|Proceedings of the National Academy of Sciences|1998|1.9k
Epigenetic Determinants of Cancer
|Cold Spring Harbor Perspectives in Biology|2016|1.3k
Evolution of Neoantigen Landscape during Immune Checkpoint Blockade in Non–Small Cell Lung Cancer
|Cancer Discovery|2016|909