Neuron-specific PERK inactivation exacerbates neurodegeneration during experimental autoimmune encephalomyelitis

Sarrabeth Stone(University of Minnesota Medical Center), Wensheng Lin(University of Chicago)

JCI Insight

January 23, 2019

10.1172/jci.insight.124232

Cited by 32

Related Papers

The integrated stress response prevents demyelination by protecting oligodendrocytes against immune-mediated damage

|Journal of Clinical Investigation|2007|198

Oligodendrocyte-Specific Activation of PERK Signaling Protects Mice against Experimental Autoimmune Encephalomyelitis

|Journal of Neuroscience|2013|111

Enhanced Integrated Stress Response Promotes Myelinating Oligodendrocyte Survival in Response to Interferon-γ

|American Journal Of Pathology|2008|101

Interferon-γ Activates Nuclear Factor-κ B in Oligodendrocytes through a Process Mediated by the Unfolded Protein Response

|PLoS ONE|2012|61

Impaired Eukaryotic Translation Initiation Factor 2B Activity Specifically in Oligodendrocytes Reproduces the Pathology of Vanishing White Matter Disease in Mice

|Journal of Neuroscience|2014|52