Cellular uptake of paraquat determines subsequent toxicity including mitochondrial damage in lung epithelial cells
Sanae Kanno(Nagoya City University), Yasuhiro Aoki(Tokyo Medical Center), Toshiji Mukai(Kobe University), Mamiko Fukuta(Nagoya City University), Ayako Ro(Tokai University), Seishiro Hirano(National Institute for Environmental Studies), Hideaki Kato(Yokohama City University Hospital)
Cited by 29
Related Papers
A new metabolic pathway of arsenite: arsenic?glutathione complexes are substrates for human arsenic methyltransferase Cyt19
|Archives of Toxicology|2004|530
Metabolism of arsenic and its toxicological relevance
|Archives of Toxicology|2012|328
Structure of insect chitin isolated from beetle larva cuticle and silkworm (Bombyx mori) pupa exuvia
|International Journal of Biological Macromolecules|2000|270
Oxidative damage to mitochondria is a preliminary step to caspase-3 activation in fluoride-induced apoptosis in HL-60 cells
|Free Radical Biology and Medicine|2001|232