Excessive tubulin polyglutamylation causes neurodegeneration and perturbs neuronal transport
Maria M. Magiera(Centre National de la Recherche Scientifique), Carsten Janke(Université Paris-Sud), Marc Landry(Centre National de la Recherche Scientifique), Annie Andrieux(Inserm), Torben J. Hausrat(University Medical Center Hamburg-Eppendorf), Satish Bodakuntla(Université Paris-Sud), Christophe Bosc(Inserm), Jakub Žiak(Johns Hopkins University), Matthias Kneussel(Universität Hamburg), Sophie Leboucher(Université Paris-Sud), Sabrina Lacomme(Centre National de la Recherche Scientifique), A. Calas(Centre National de la Recherche Scientifique), Patricia Marques Sousa(Université Paris-Sud), Martin Balaštík(Czech Academy of Sciences)
Cited by 157
Related Papers
Radixin regulates synaptic GABAA receptor density and is essential for reversal learning and short-term memory
|Nature Communications|2015|139
Trim17-mediated ubiquitination and degradation of Mcl-1 initiate apoptosis in neurons
|Cell Death and Differentiation|2012|103
Mitochondrial function in skeletal muscle of patients with protracted critical illness and ICU-acquired weakness
|Critical Care|2015|72
Microtubule-associated protein 6 mediates neuronal connectivity through Semaphorin 3E-dependent signalling for axonal growth
|Nature Communications|2015|65
Neuroligin 1 Is Dynamically Exchanged at Postsynaptic Sites
|Journal of Neuroscience|2010|63