Mitochondrial dysfunction contributes to the senescent phenotype of <scp>IPF</scp> lung fibroblasts
Michael Schuliga(University of Newcastle Australia), Darryl A. Knight(Providence Health Care Research Institute)
Cited by 87
Related Papers
Transforming Growth Factor β1 Induces αvβ3 Integrin Expression in Human Lung Fibroblasts via a β3 Integrin-, c-Src-, and p38 MAPK-dependent Pathway
|Journal of Biological Chemistry|2008|98
Antifibrotic role of vascular endothelial growth factor in pulmonary fibrosis
|JCI Insight|2017|85
STAT3 Regulates the Onset of Oxidant-induced Senescence in Lung Fibroblasts
|American Journal of Respiratory Cell and Molecular Biology|2019|70
A confocal microscopic study of solitary pulmonary neuroendocrine cells in human airway epithelium
|Respiratory Research|2005|60
Self DNA perpetuates IPF lung fibroblast senescence in a cGAS-dependent manner
|Clinical Science|2020|55