Length-independent telomere damage drives cardiomyocyte senescence

Rhys Anderson(Newcastle Hospitals - Campus for Ageing and Vitality), João F. Passos(Newcastle University), Peter D. Adams(Discovery Institute), Neil Robertson(University of Glasgow), W. Andrew Owens(Newcastle University), Jodie Birch(Newcastle University), Laura C. Greaves(Queen Mary University of London), James L. Kirkland(Cedars-Sinai Medical Center), Diana Jurk(Mayo Clinic in Florida), Clara Correia‐Melo(Leibniz Institute on Aging - Fritz Lipmann Institute (FLI)), Nathan K. LeBrasseur(WinnMed), Simon Tual‐Chalot(Newcastle University), Tamar Tchkonia(Cedars-Sinai Medical Center), Edward Fielder(Newcastle University), Helen M. Arthur(Newcastle University), Rolando Berlinguer‐Palmini(Newcastle University), James Chapman(George Washington University), Jeanne Mialet‐Perez(Centre National de la Recherche Scientifique), Stella Victorelli(Mayo Clinic in Florida), Damien Maggiorani(Centre Hospitalier Universitaire Sainte-Justine), Hanna Salmonowicz(Mayo Clinic in Florida), Angelo Parini(Inserm), Jordan D. Miller(Queen's University), Victorine Douin‐Echinard(Inserm), Gavin D. Richardson(Newcastle University), Mikołaj Ogrodnik(Austrian Cluster for Tissue Regeneration), Anthony B. Lagnado(Mayo Clinic), Emily Dookun(Newcastle University), Carolyn M Roos(Amsterdam UMC Location University of Amsterdam), Anna Walaszczyk(Newcastle University), Jelena Mann(Newcastle University), Carole J. Proctor(Newcastle Hospitals - Campus for Ageing and Vitality), Marissa Schafer(WinnMed), Kathy L. Kolsky(Mayo Clinic in Florida)
bioRxiv (Cold Spring Harbor Laboratory)
August 17, 2018
Cited by 6


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