Mitochondrial dysfunction in pathophysiology of heart failure
Cited by 909
Related Papers
Cardiac-Specific Expression of Heme Oxygenase-1 Protects Against Ischemia and Reperfusion Injury in Transgenic Mice
|Circulation Research|2001|419
Cardiac hypertrophy with preserved contractile function after selective deletion of GLUT4 from the heart
|Journal of Clinical Investigation|1999|364
Decreased Contractile and Metabolic Reserve in Peroxisome Proliferator–Activated Receptor-α–Null Hearts Can Be Rescued by Increasing Glucose Transport and Utilization
|Circulation|2005|175
Deletion of thioredoxin-interacting protein in mice impairs mitochondrial function but protects the myocardium from ischemia-reperfusion injury
|Journal of Clinical Investigation|2011|157
Glucose promotes cell growth by suppressing branched-chain amino acid degradation
|Nature Communications|2018|152