Long-Acting β2-Adrenoceptor Agonists Enhance Glucocorticoid Receptor (GR)–Mediated Transcription by Gene-Specific Mechanisms Rather Than Generic Effects via GR
Christopher F. Rider(University of Calgary), Robert Newton(University of Calgary)
Cited by 31
Related Papers
Long-acting β<sub>2</sub>-agonists promote glucocorticoid-mediated repression of NF-κB by enhancing expression of the feedback regulator TNFAIP3
|American Journal of Physiology-Lung Cellular and Molecular Physiology|2016|44
Cytokine-Induced Loss of Glucocorticoid Function: Effect of Kinase Inhibitors, Long-Acting β2-Adrenoceptor Agonist and Glucocorticoid Receptor Ligands
|PLoS ONE|2015|30
DUSP1 Maintains IRF1 and Leads to Increased Expression of IRF1-dependent Genes
|Journal of Biological Chemistry|2016|29
Differential regulation of BIRC2 and BIRC3 expression by inflammatory cytokines and glucocorticoids in pulmonary epithelial cells
|PLoS ONE|2023|17
Modulation of glucocorticoid-inducible gene expression: Effects of inflammatory stimuli and long-acting β2-adrenoceptor agonists
|PRISM (University of Calgary)|2014|0