Abstract 4676: Spontaneous regression of Merkel cell carcinoma is driven by adaptive immune activation and clonal T cell expansion
Mairead Baker(National Institutes of Health), Isaac Brownell(National Institute of Arthritis and Musculoskeletal and Skin Diseases), John W Roman(Walter Reed National Military Medical Center), Natasha T. Hill(National Institutes of Health), Alexandre Reuben(The University of Texas MD Anderson Cancer Center), Nicholas Logemann(Walter Reed National Military Medical Center), Michael T. Tetzlaff(Hospital of the University of Pennsylvania), Courtney W. Hudgens(The University of Texas MD Anderson Cancer Center)
Cited by 3
Related Papers
Defining T Cell States Associated with Response to Checkpoint Immunotherapy in Melanoma
|Cell|2018|2k
Potential role of intratumor bacteria in mediating tumor resistance to the chemotherapeutic drug gemcitabine
|Science|2017|1.8k
Dietary fiber and probiotics influence the gut microbiome and melanoma immunotherapy response
|Science|2021|876
Pan-cancer T cell atlas links a cellular stress response state to immunotherapy resistance
|Nature Medicine|2023|508
The Abscopal Effect Associated With a Systemic Anti-melanoma Immune Response
|International Journal of Radiation Oncology*Biology*Physics|2013|380