Abstract 15515: Mechanism of Fever-Induced QT Prolongation in Patients With KCNH2 Mutations in the S5-Pore Region: Evidence From Genotypic and Functional Analyses

Kenshi Hayashi(Kanazawa University), Masakazu Yamagishi, Noboru Fujino(Kanazawa University), Yoshiaki Kaneko(Cardiovascular Institute Hospital), Yoji Nagata(Fukui CardioVascular Center), Tadashi Nakajima(Gunma University), Ryota Teramoto(Ishikawa Prefectural Central Hospital), Masa‐aki Kawashiri(Kanazawa University), Shoichi Tange(Maebashi Red Cross Hospital), Yasutaka Kurata(Kanazawa Medical University), Masahiko Kurabayashi(Gunma University), Kenji Sakata(Kanazawa University), Takekatsu Saito(Kanazawa University), Tetsuo Konno(Kanazawa University), Yoshihiro Tanaka(Kanazawa University), Toyonobu Tsuda(Kanazawa University), Kunio Ohta(Kanazawa University)
Circulation
November 11, 2016
Cited by 0

Related Papers

Cardiac fibrosis in mice with hypertrophic cardiomyopathy is mediated by non-myocyte proliferation and requires Tgf-β
|Journal of Clinical Investigation|2010|440
T wave peak-to-end interval and QT dispersion in acquired long QT syndrome: a new index for arrhythmogenicity
|Clinical Science|2003|313
T‐peak to T‐end interval may be a better predictor of high‐risk patients with hypertrophic cardiomyopathy associated with a cardiac troponin i mutation than qt dispersion
|Clinical Cardiology|2002|267
Impact of clinical signs and genetic diagnosis of familial hypercholesterolaemia on the prevalence of coronary artery disease in patients with severe hypercholesterolaemia
|European Heart Journal|2017|165
Lumen Loss in Transplant Coronary Artery Disease Is a Biphasic Process Involving Early Intimal Thickening and Late Constrictive Remodeling
|Circulation|2001|163