Autocrine Tnf signaling favors malignant cells in myelofibrosis in a Tnfr2-dependent fashion

William L. Heaton(University of Utah), Michael W. Deininger(Versiti Blood Center of Wisconsin), Mohamed E. Salama(The University of Texas at San Antonio Health Science Center), Josef T. Prchal(Huntsman Cancer Institute), Jeremy M. Gililland(University of Utah), Anna V. Senina(Astex Pharmaceuticals), Dongqing Yan(Sun Yat-sen University), Russell Bell(University of Utah), Anthony D. Pomicter(University of Utah), Thomas O’Hare(University of Utah), Phillip M. Clair(University of Utah)
Leukemia
April 18, 2018
10.1038/s41375-018-0131-z
Cited by 51

Related Papers

MYC Drives Progression of Small Cell Lung Cancer to a Variant Neuroendocrine Subtype with Vulnerability to Aurora Kinase Inhibition
|Cancer Cell|2017|619
Spectrum of Epithelial Neoplasms in End-Stage Renal Disease
|The American Journal of Surgical Pathology|2006|484
Asciminib in Chronic Myeloid Leukemia after ABL Kinase Inhibitor Failure
|New England Journal of Medicine|2019|464
Low-dose total body irradiation (TBI) and fludarabine followed by hematopoietic cell transplantation (HCT) from HLA-matched or mismatched unrelated donors and postgrafting immunosuppression with cyclosporine and mycophenolate mofetil (MMF) can induce durable complete chimerism and sustained remissions in patients with hematological diseases
|Blood|2003|457
A genetic mechanism for Tibetan high-altitude adaptation
|Nature Genetics|2014|441