Innate immune adaptor TRIF deficiency accelerates disease progression of ALS mice with accumulation of aberrantly activated astrocytes

Okiru Komine(Nagoya University), Koji Yamanaka(RIKEN Center for Brain Science)
Cell Death and Differentiation
March 22, 2018
10.1038/s41418-018-0098-3
Cited by 57

Related Papers

Mitochondria‐associated membrane collapse is a common pathomechanism in SIGMAR1‐ and SOD1‐linked ALS
|EMBO Molecular Medicine|2016|222
SIRT1 overexpression ameliorates a mouse model of SOD1-linked amyotrophic lateral sclerosis via HSF1/HSP70i chaperone system
|Molecular Brain|2014|91
Cystatin C protects neuronal cells against mutant copper-zinc superoxide dismutase-mediated toxicity
|Cell Death and Disease|2014|79
Monomerization of TDP-43 is a key determinant for inducing TDP-43 pathology in amyotrophic lateral sclerosis
|Science Advances|2023|64
Aggresome formation and liquid–liquid phase separation independently induce cytoplasmic aggregation of TAR DNA-binding protein 43
|Cell Death and Disease|2020|55