Enhancer redundancy provides phenotypic robustness in mammalian development

Marco Osterwalder(Lawrence Berkeley National Laboratory), Iros Barozzi(Lawrence Berkeley National Laboratory), Virginie Tissières(University of Basel), Y. Fukuda(Lawrence Berkeley National Laboratory), Brandon J. Mannion(Lawrence Berkeley National Laboratory), Sarah Afzal(Lawrence Berkeley National Laboratory), Elizabeth A. Lee(Lawrence Berkeley National Laboratory), Yiwen Zhu(Lawrence Berkeley National Laboratory), Ingrid Plajzer-Frick(Lawrence Berkeley National Laboratory), Catherine S. Pickle(Lawrence Berkeley National Laboratory), Momoe Kato(Lawrence Berkeley National Laboratory), Tyler H. Garvin(Lawrence Berkeley National Laboratory), Quan Pham(Lawrence Berkeley National Laboratory), Anne Harrington(Lawrence Berkeley National Laboratory), Jennifer A. Akiyama(Lawrence Berkeley National Laboratory), Veena Afzal(Lawrence Berkeley National Laboratory), Javier López-Rı́os(University of Basel), Diane E. Dickel(Lawrence Berkeley National Laboratory), Axel Visel(Lawrence Berkeley National Laboratory), L Pennacchio(Joint Genome Institute)
Nature
January 31, 2018
Cited by 707Open Access
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