Tumor necrosis factor-α acts reciprocally with solute carrier family 26, member 3, (downregulated-in-adenoma) and reduces its expression, leading to intestinal inflammation
Xiangming Ding(Tongji Hospital), Qin Yu(Chongqing Jiaotong University)
Cited by 24
Related Papers
The role of Lactobacillus in inflammatory bowel disease: from actualities to prospects
|Cell Death Discovery|2023|184
Signaling between Transforming Growth Factor β (TGF-β) and Transcription Factor SNAI2 Represses Expression of MicroRNA miR-203 to Promote Epithelial-Mesenchymal Transition and Tumor Metastasis
|Journal of Biological Chemistry|2013|163
Butyrate Inhibits the HDAC8/NF-κB Pathway to Enhance Slc26a3 Expression and Improve the Intestinal Epithelial Barrier to Relieve Colitis
|Journal of Agricultural and Food Chemistry|2024|60
Short‐chain fatty acids affect the development of inflammatory bowel disease through intestinal barrier, immunology, and microbiota: A promising therapy?
|Journal of Gastroenterology and Hepatology|2022|58
The role of sphingosine-1-phosphate in autophagy and related disorders
|Cell Death Discovery|2023|40