Reduced cell size, chromosomal aberration and altered proliferation rates are characteristics and confounding factors in the STHdh cell model of Huntington disease
Elisabeth Singer(University of Tübingen), Huu Phuc Nguyen(University of Tübingen), Olaf Rieß(STZ eyetrial), Nadine Rischert(University of Tübingen), Laura E. Clemensson(Uppsala University), Ann-Christin Krahl(University Children's Hospital Tübingen), Carolin Walter(University of Tübingen), Ulrike A. Mau‐Holzmann(University of Tübingen), Jonasz Jeremiasz Weber(University Hospitals of the Ruhr-University of Bochum)
Cited by 21
Related Papers
Calpain-mediated ataxin-3 cleavage in the molecular pathogenesis of spinocerebellar ataxia type 3 (SCA3)
|Human Molecular Genetics|2012|78
From Pathways to Targets: Understanding the Mechanisms behind Polyglutamine Disease
|BioMed Research International|2014|70
Dynamics of cell-free tumour DNA correlate with treatment response of head and neck cancer patients receiving radiochemotherapy
|Radiotherapy and Oncology|2020|57
A shift in the collagen V antigenic epitope leads to T helper phenotype switch and immune response to self-antigen leading to chronic lung allograft rejection
|Clinical & Experimental Immunology|2011|56
Decreased iron absorption in patients with active rheumatoid arthritis, with and without iron deficiency.
|Annals of the Rheumatic Diseases|1988|56