Engineered D2R Variants Reveal the Balanced and Biased Contributions of G-Protein and β-Arrestin to Dopamine-Dependent Functions
Samuel J. Rose(Duke Medical Center), Marc G. Caron(Howard Hughes Medical Institute)
Cited by 29
Related Papers
DESENSITIZATION OF G PROTEIN–COUPLED RECEPTORS AND NEURONAL FUNCTIONS
|Annual Review of Neuroscience|2004|859
MicroRNA-219 modulates NMDA receptor-mediated neurobehavioral dysfunction
|Proceedings of the National Academy of Sciences|2009|299
Differential mechanisms of morphine antinociceptive tolerance revealed in (beta)arrestin-2 knock-out mice.
|PubMed|2002|268
Regulation of Akt Signaling by D<sub>2</sub>and D<sub>3</sub>Dopamine Receptors<i>In Vivo</i>
|Journal of Neuroscience|2007|265
Differential Mechanisms of Morphine Antinociceptive Tolerance Revealed in βArrestin-2 Knock-Out Mice
|Journal of Neuroscience|2002|243