Keap1 loss promotes Kras-driven lung cancer and results in dependence on glutaminolysis
Rodrigo Romero(Massachusetts Institute of Technology), Volkan I. Sayin(New York University), Shawn M. Davidson(Massachusetts Institute of Technology), Matthew R. Bauer(Massachusetts Institute of Technology), Simranjit X. Singh(New York University), Sarah E. LeBoeuf(New York University), Triantafyllia Karakousi(New York University), Donald C Ellis(Massachusetts Institute of Technology), Arjun Bhutkar(Massachusetts Institute of Technology), Francisco J. Sánchez‐Rivera(Massachusetts Institute of Technology), Lakshmipriya Subbaraj(Massachusetts Institute of Technology), Britney Martinez(New York University), Roderick T. Bronson(Tufts University), Justin R. Prigge(Montana State University), Edward E. Schmidt(Montana State University), Craig J. Thomas(National Institutes of Health), Chandra Goparaju(NYU Langone Health), Angela M. Davies(Champions Oncology (United States)), Igor Dolgalev(New York Genome Center), Adriana Heguy(New York Genome Center), Viola Allaj(Memorial Sloan Kettering Cancer Center), John T. Poirier(Memorial Sloan Kettering Cancer Center), André L. Moreira(New York University), Charles M. Rudin(Memorial Sloan Kettering Cancer Center), Harvey I. Pass(NYU Langone Health), Matthew G. Vander Heiden(Massachusetts Institute of Technology), Tyler Jacks(Howard Hughes Medical Institute), Thales Papagiannakopoulos(New York University)
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