TRAF3IP2 mediates high glucose-induced endothelin-1 production as well as endothelin-1-induced inflammation in endothelial cells
Jaume Padilla(Harry S. Truman Memorial Veterans' Hospital), Bysani Chandrasekar(University of Missouri)
American Journal of Physiology-Heart and Circulatory Physiology
October 2, 2017
Cited by 37
Related Papers
Interleukin-18-induced Human Coronary Artery Smooth Muscle Cell Migration Is Dependent on NF-κB- and AP-1-mediated Matrix Metalloproteinase-9 Expression and Is Inhibited by Atorvastatin
|Journal of Biological Chemistry|2006|194
Interleukin-17 Stimulates C-reactive Protein Expression in Hepatocytes and Smooth Muscle Cells via p38 MAPK and ERK1/2-dependent NF-κB and C/EBPβ Activation
|Journal of Biological Chemistry|2007|191
Fractalkine (CX3CL1) stimulated by nuclear factor kappaB (NF-kappaB)-dependent inflammatory signals induces aortic smooth muscle cell proliferation through an autocrine pathway
|Biochemical Journal|2003|157
CXCL16 Signals via Gi, Phosphatidylinositol 3-Kinase, Akt, IκB Kinase, and Nuclear Factor-κB and Induces Cell-Cell Adhesion and Aortic Smooth Muscle Cell Proliferation
|Journal of Biological Chemistry|2004|157
Neutralization of Interleukin-18 Ameliorates Ischemia/Reperfusion-induced Myocardial Injury
|Journal of Biological Chemistry|2009|131