Combined fetal inflammation and postnatal hypoxia causes myelin deficits and autism‐like behavior in a rat model of diffuse white matter injury

Erik van Tilborg(University Medical Center Utrecht), Cora H. Nijboer(Wilhelmina Children's Hospital)

Glia

September 19, 2017

10.1002/glia.23216

Cited by 84

Related Papers

Low Endogenous G-Protein-Coupled Receptor Kinase 2 Sensitizes the Immature Brain to Hypoxia–Ischemia-Induced Gray and White Matter Damage

|Journal of Neuroscience|2008|32

Timed fetal inflammation and postnatal hypoxia cause cortical white matter injury, interneuron imbalances, and behavioral deficits in a double-hit rat model of encephalopathy of prematurity

|Brain Behavior & Immunity - Health|2024|6