Rapamycin-induced miR-21 promotes mitochondrial homeostasis and adaptation in mTORC1 activated cells
Hilaire C. Lam(Brigham and Women's Hospital), Elizabeth P. Henske(Brigham and Women's Hospital), Alicia Llorente Lope(Brigham and Women's Hospital), Heng Du(Brigham and Women's Hospital), Stephen Y. Chan(University of Pittsburgh Medical Center), Harilaos Filippakis(Brigham and Women's Hospital), Issam Ben‐Sahra(Northwestern University), John M. Asara(Beth Israel Deaconess Medical Center), Christian Baglini(Brigham and Women's Hospital), Heng-Jia Liu(Brigham and Women's Hospital), Julie Nijmeh(Brigham and Women's Hospital), Adam Handen(University of Pittsburgh), William M. Oldham(Brigham and Women's Hospital), Katherine A. Cottrill(University of Pittsburgh Medical Center), David J. Kwiatkowski(Brigham and Women's Hospital), Nicola Alesi(Brigham and Women's Hospital)
Cited by 23
Related Papers
Autophagy proteins regulate innate immune responses by inhibiting the release of mitochondrial DNA mediated by the NALP3 inflammasome
|Nature Immunology|2010|3k
Oncogenic Kras Maintains Pancreatic Tumors through Regulation of Anabolic Glucose Metabolism
|Cell|2012|2k
Phosphoglycerate dehydrogenase diverts glycolytic flux and contributes to oncogenesis
|Nature Genetics|2011|1.1k
Vitamin C selectively kills <i>KRAS</i> and <i>BRAF</i> mutant colorectal cancer cells by targeting GAPDH
|Science|2015|907
The Somatic Genomic Landscape of Chromophobe Renal Cell Carcinoma
|Cancer Cell|2014|860