HGFL-mediated RON signaling supports breast cancer stem cell phenotypes via activation of non-canonical β-catenin signalingSasha J. Ruiz-Torres(University of Cincinnati Medical Center), Susan E. Waltz(Veterans Health Administration)OncotargetJuly 22, 201710.18632/oncotarget.19441Cited by 23SaveCiteExport RISWatch citationsRelated PapersMammary-Specific Ron Receptor Overexpression Induces Highly Metastatic Mammary Tumors Associated with β-Catenin Activation|Cancer Research|2006|102HGFL supports mammary tumorigenesis by enhancing tumor cell intrinsic survival and influencing macrophage and T-cell responses|Oncotarget|2015|17Hepatocyte growth factor-like protein is required for prostate tumor growth in the TRAMP mouse model|Oncotarget|2014|12