BAP1 regulates IP3R3-mediated Ca2+ flux to mitochondria suppressing cell transformation

Angela Bononi(University of Hawaii Cancer Center), Carlotta Giorgi(University of Ferrara), Simone Patergnani(University of Ferrara), David Larson(University of Hawaii Cancer Center), Kaitlyn Verbruggen(University of Hawaiʻi at Mānoa), Mika Tanji(University of Hawaii Cancer Center), Laura Pellegrini(University of Hawaiʻi at Mānoa), Valentina Signorato(University of Hawaii System), Federica Olivetto(University of Hawaii Cancer Center), Sandra Pastorino(University of Hawaii System), Masaki Nasu(University of Hawaii System), Andrea Napolitano(University of Hawaii Cancer Center), Giovanni Gaudino(University of Hawaiʻi at Mānoa), Paul Morris(University of Hawaii System), G Sakamoto(University of Hawaiʻi at Mānoa), Laura K. Ferris(University of Pittsburgh Medical Center), Alberto Danese(University of Ferrara), Andrea Raimondi(San Raffaele University of Rome), Carlo Tacchetti(San Raffaele University of Rome), Shafi Kuchay(New York University), Harvey I. Pass(New York University), El Bachir Affar(Hôpital Maisonneuve-Rosemont), Haining Yang(University of Hawaii Cancer Center), Paolo Pinton(University of Ferrara), Michele Carbone(University of Hawaii Cancer Center)
Nature
June 1, 2017
10.1038/nature22798
Cited by 398Open Access
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