Interaction between autophagy and senescence is required for dihydroartemisinin to alleviate liver fibrosis

Zili Zhang(Nanjing University of Chinese Medicine), Shizhong Zheng(Nanjing University of Chinese Medicine)
Cell Death and Disease
June 15, 2017
10.1038/cddis.2017.255
Cited by 96

Related Papers

Activation of PPARγ/P53 signaling is required for curcumin to induce hepatic stellate cell senescence
|Cell Death and Disease|2016|139
Curcumin attenuates angiogenesis in liver fibrosis and inhibits angiogenic properties of hepatic stellate cells
|Journal of Cellular and Molecular Medicine|2014|133
RETRACTED: Autophagy regulates turnover of lipid droplets via ROS-dependent Rab25 activation in hepatic stellate cell
|Redox Biology|2016|105
Tetramethylpyrazine reduces inflammation in liver fibrosis and inhibits inflammatory cytokine expression in hepatic stellate cells by modulating <scp>NLRP</scp>3 inflammasome pathway
|IUBMB Life|2015|89
Curcumin attenuates ethanol‐induced hepatic steatosis through modulating <scp>N</scp>rf2/<scp>FXR</scp> signaling in hepatocytes
|IUBMB Life|2015|88