Human LACC1 increases innate receptor-induced responses and a LACC1 disease-risk variant modulates these outcomes
Amit Lahiri(Central Drug Research Institute), Clara Abraham(Yale University)
Cited by 75
Related Papers
Host–microbe interactions have shaped the genetic architecture of inflammatory bowel disease
|Nature|2012|4.8k
A Genome-Wide Association Study Identifies <i>IL23R</i> as an Inflammatory Bowel Disease Gene
|Science|2006|3k
Inflammatory Bowel Disease
|New England Journal of Medicine|2009|2.7k
IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine
|Nature|2012|705
Ulcerative colitis–risk loci on chromosomes 1p36 and 12q15 found by genome-wide association study
|Nature Genetics|2009|393