Illuminating the leukemogenic transformation activity of TCF3-HLF in pediatric ALL

Brice Mouttet(University Children's Hospital Zurich), Jean‐Pierre Bourquin(University Children's Hospital Zurich), Beat Bornhäuser(University Children's Hospital Zurich), HJ Warnatz(Max Planck Institute for Molecular Genetics), Yü Huang(University of California, Los Angeles), ML Yaspo(Max Planck Institute for Molecular Genetics)
Klinische Pädiatrie
May 1, 2017
10.1055/s-0037-1602217
Cited by 0

Related Papers

Down syndrome acute lymphoblastic leukemia, a highly heterogeneous disease in which aberrant expression of CRLF2 is associated with mutated JAK2: a report from the International BFM Study Group
|Blood|2009|347
Induction of autophagy-dependent necroptosis is required for childhood acute lymphoblastic leukemia cells to overcome glucocorticoid resistance
|Journal of Clinical Investigation|2010|311
<i>IKZF1</i><sup>plus</sup>Defines a New Minimal Residual Disease–Dependent Very-Poor Prognostic Profile in Pediatric B-Cell Precursor Acute Lymphoblastic Leukemia
|Journal of Clinical Oncology|2018|304
Efficient apoptosis requires feedback amplification of upstream apoptotic signals by effector caspase-3 or -7
|Science Advances|2019|274
Ex vivo drug response profiling detects recurrent sensitivity patterns in drug-resistant acute lymphoblastic leukemia
|Blood|2017|263