Randomized Trial of Irinotecan and Cetuximab With or Without Vemurafenib in BRAF-Mutant Metastatic Colorectal Cancer (SWOG S1406)

Scott Kopetz(The University of Texas MD Anderson Cancer Center), Katherine A. Guthrie, Van K. Morris(The University of Texas MD Anderson Cancer Center), Heinz‐Josef Lenz(University of Southern California), Anthony M. Magliocco(Moffitt Cancer Center), Dipen M. Maru(The University of Texas MD Anderson Cancer Center), Yibing Yan, Richard B. Lanman(Guardant (United States)), Ganiraju C. Manyam(The University of Texas MD Anderson Cancer Center), David S. Hong(The University of Texas MD Anderson Cancer Center), Alexey V. Sorokin(The University of Texas MD Anderson Cancer Center), Chloé E. Atreya(University of California, San Francisco), Luis A. Díaz(Memorial Sloan Kettering Cancer Center), Carmen J. Allegra(University of Florida), Kanwal Raghav(The University of Texas MD Anderson Cancer Center), Stephen E Wang(Kaiser Permanente South Sacramento Medical Center), Christopher H. Lieu(University of Colorado Denver), Shannon McDonough, Philip A. Philip(Wayne State University), Howard S. Höchster(Rutgers, The State University of New Jersey)
Journal of Clinical Oncology
December 23, 2020
Cited by 390Open Access
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Abstract

PURPOSE BRAF V600E mutations are rarely associated with objective responses to the BRAF inhibitor vemurafenib in patients with metastatic colorectal cancer (CRC). Blockade of BRAF V600E by vemurafenib causes feedback upregulation of EGFR, whose signaling activities can be impeded by cetuximab. METHODS One hundred six patients with BRAF V600E -mutated metastatic CRC previously treated with one or two regimens were randomly assigned to irinotecan and cetuximab with or without vemurafenib (960 mg PO twice daily). RESULTS Progression-free survival, the primary end point, was improved with the addition of vemurafenib (hazard ratio, 0.50, P = .001). The response rate was 17% versus 4% ( P = .05), with a disease control rate of 65% versus 21% ( P < .001). A decline in circulating tumor DNA BRAF V600E variant allele frequency was seen in 87% versus 0% of patients ( P < .001), with a low incidence of acquired RAS alterations at the time of progression. RNA profiling suggested that treatment benefit did not depend on previously established BRAF subgroups or the consensus molecular subtype. CONCLUSION Simultaneous inhibition of EGFR and BRAF combined with irinotecan is effective in BRAF V600E -mutated CRC.


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