Ubiquitylation of p62/sequestosome1 activates its autophagy receptor function and controls selective autophagy upon ubiquitin stress
Hong Peng(Chinese Academy of Sciences), Jiao Yang(Chinese Academy of Sciences), Guangyi Li(Center for Excellence in Molecular Cell Science), Qing You(Center for Excellence in Molecular Cell Science), Wen Han(Center for Excellence in Molecular Cell Science), Tianrang Li(Center for Excellence in Molecular Cell Science), Daming Gao(Center for Excellence in Molecular Cell Science), Xiaoduo Xie(Center for Excellence in Molecular Cell Science), Byung‐Hoon Lee(Harvard University), Juan Du(Second Military Medical University), Jian Hou(Second Military Medical University), Tao Zhang(Fudan University), Hai Rao(The University of Texas at San Antonio Health Science Center), Ying Huang(Chinese Academy of Sciences), Qingrun Li(Center for Excellence in Molecular Cell Science), Rong Zeng(Center for Excellence in Molecular Cell Science), Lijian Hui(Chinese Academy of Sciences), Hongyan Wang(Center for Excellence in Molecular Cell Science), Xia Qin(National Center of Biomedical Analysis), Xuemin Zhang(National Center of Biomedical Analysis), Yongning He(Chinese Academy of Sciences), Masaaki Komatsu(Niigata University), Ivan Đikić(Goethe University Frankfurt), Daniel Finley(Harvard University), Ronggui Hu(Center for Excellence in Molecular Cell Science)
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