3D pulmospheres serve as a personalized and predictive multicellular model for assessment of antifibrotic drugs
Ranu Surolia(Pulmonary and Allergy Associates), Veena B. Antony(Pulmonary and Allergy Associates), Huashi Li(Pulmonary and Allergy Associates), Tracy Luckhardt(University of Alabama at Birmingham), Zheng Wang(Xidian University), Sunad Rangarajan(University of Alabama at Birmingham), Victor J. Thannickal(Pulmonary and Allergy Associates), Yong Zhou(Tulane University), Gang Liu(Tianjin Huanhu Hospital), Sejong Bae(University of Alabama at Birmingham), Fu Jun Li(University of Alabama at Birmingham), Rui-Ming Liu(University of Alabama at Birmingham), João A. de Andrade(University of Alabama at Birmingham)
Cited by 61
Related Papers
miR-21 mediates fibrogenic activation of pulmonary fibroblasts and lung fibrosis
|The Journal of Experimental Medicine|2010|931
Allosteric inhibition of SHP2 phosphatase inhibits cancers driven by receptor tyrosine kinases
|Nature|2016|925
Matrix Stiffness–Induced Myofibroblast Differentiation Is Mediated by Intrinsic Mechanotransduction
|American Journal of Respiratory Cell and Molecular Biology|2012|527
Inhibition of mechanosensitive signaling in myofibroblasts ameliorates experimental pulmonary fibrosis
|Journal of Clinical Investigation|2013|472
What's in a name? That which we call IPF, by any other name would act the same
|European Respiratory Journal|2018|352