The Zn 2+ -sensing receptor, ZnR/GPR39, upregulates colonocytic Cl − absorption, via basolateral KCC1, and reduces fluid loss
Laxmi Sunuwar(Ben-Gurion University of the Negev), Michal Hershfinkel(Ben-Gurion University of the Negev), Mark Donowitz(Johns Hopkins University), Hila Asraf(Ben-Gurion University of the Negev), Israel Sekler(Ben-Gurion University of the Negev)
Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease
January 16, 2017
Cited by 32
Related Papers
NCLX is an essential component of mitochondrial Na <sup>+</sup> /Ca <sup>2+</sup> exchange
|Proceedings of the National Academy of Sciences|2009|776
Zinc in the physiology and pathology of the CNS
|Nature reviews. Neuroscience|2009|759
A zinc-sensing receptor triggers the release of intracellular Ca <sup>2+</sup> and regulates ion transport
|Proceedings of the National Academy of Sciences|2001|257
Synaptically Released Zinc Triggers Metabotropic Signaling via a Zinc-Sensing Receptor in the Hippocampus
|Journal of Neuroscience|2009|229
Role of GPR40 in fatty acid action on the β cell line INS-1E
|Biochemical and Biophysical Research Communications|2005|222