Translational upregulation of Aurora-A by hnRNP Q1 contributes to cell proliferation and tumorigenesis in colorectal cancer

Chien-Hsien Lai(National Cheng Kung University), Yu-Chuan Huang(National Cheng Kung University), Jenq-Chang Lee(National Cheng Kung University Hospital), Joseph T. Tseng(National Cheng Kung University), Kung‐Chao Chang(National Cheng Kung University Hospital), Yen‐Ju Chen(National Cheng Kung University), Nai-Jhu Ding(National Cheng Kung University), Pao-Hsuan Huang(National Cheng Kung University), Wen‐Chang Chang(Taipei Medical University), Bo‐Wen Lin(National Cheng Kung University Hospital), Ruo-Yu Chen(National Cheng Kung University), Yu-Chu Wang(National Cheng Kung University), Yi-Chien Lai(National Cheng Kung University), Liang‐Yi Hung(Taipei Medical University)
Cell Death and Disease
January 12, 2017
Cited by 47Open Access
Full Text

Abstract

By using RNA-immunoprecipitation assay following next-generation sequencing, a group of cell cycle-related genes targeted by hnRNP Q1 were identified, including Aurora-A kinase. Overexpressed hnRNP Q1 can upregulate Aurora-A protein, but not alter the mRNA level, through enhancing the translational efficiency of Aurora-A mRNA, either in a cap-dependent or -independent manner, by interacting with the 5'-UTR of Aurora-A mRNA through its RNA-binding domains (RBDs) 2 and 3. By ribosomal profiling assay further confirmed the translational regulation of Aurora-A mRNA by hnRNP Q1. Overexpression of hnRNP Q1 promotes cell proliferation and tumor growth. HnRNP Q1/ΔRBD23-truncated mutant, which loses the binding ability and translational regulation of Aurora-A mRNA, has no effect on promoting tumor growth. The expression level of hnRNP Q1 is positively correlated with Aurora-A in colorectal cancer. Taken together, our data indicate that hnRNP Q1 is a novel trans-acting factor that binds to Aurora-A mRNA 5'-UTRs and regulates its translation, which increases cell proliferation and contributes to tumorigenesis in colorectal cancer.


Related Papers

No related papers found

Powered by citation graph analysis