Angiotensin‐(1–7) protects cardiomyocytes against high glucose‐induced injuries through inhibiting reactive oxygen species‐activated leptin–p38 mitogen‐activated protein kinase/extracellular signal‐regulated protein kinase 1/2 pathways, but not the leptin–c‐Jun N‐terminal kinase pathway <i>in vitro</i>
Yiyan Lei(Sun Yat-sen University), Jingfu Chen(The First Affiliated Hospital, Sun Yat-sen University)
Cited by 26
Related Papers
Naringin Inhibits <scp>ROS</scp>‐activated <scp>MAPK</scp> Pathway in High Glucose‐induced Injuries in H9c2 Cardiac Cells
|Basic & Clinical Pharmacology & Toxicology|2013|77
Exogenous hydrogen sulfide exerts proliferation/anti-apoptosis/angiogenesis/migration effects via amplifying the activation of NF-κB pathway in PLC/PRF/5 hepatoma cells
|International Journal of Oncology|2015|72
Naringin protects cardiomyocytes against hyperglycemia-induced injuries in vitro and in vivo
|Journal of Endocrinology|2016|65
Exogenous hydrogen sulfide promotes C6 glioma cell growth through activation of the p38 MAPK/ERK1/2-COX-2 pathways
|Oncology Reports|2015|42
Inhibition of the leptin-induced activation of the p38 MAPK pathway contributes to the protective effects of naringin against high glucose-induced injury in H9c2 cardiac cells
|International Journal of Molecular Medicine|2014|35