Rats with a missense mutation in Atm display neuroinflammation and neurodegeneration subsequent to accumulation of cytosolic DNA following unrepaired DNA damage
Hazel Quek(The University of Queensland), Martin F. Lavin(Shandong First Medical University)
Cited by 49
Related Papers
DNA‐dependent protein kinase catalytic subunit: a target for an ICE‐like protease in apoptosis.
|The EMBO Journal|1996|346
Oxidative Stress Is Responsible for Deficient Survival and Dendritogenesis in Purkinje Neurons from Ataxia-Telangiectasia Mutated Mutant Mice
|Journal of Neuroscience|2003|135
ALS monocyte-derived microglia-like cells reveal cytoplasmic TDP-43 accumulation, DNA damage, and cell-specific impairment of phagocytosis associated with disease progression
|Journal of Neuroinflammation|2022|102
Aprataxin, poly-ADP ribose polymerase 1 (PARP-1) and apurinic endonuclease 1 (APE1) function together to protect the genome against oxidative damage
|Human Molecular Genetics|2009|91
ATM Protein-dependent Phosphorylation of Rad50 Protein Regulates DNA Repair and Cell Cycle Control
|Journal of Biological Chemistry|2011|91