HigB of Pseudomonas aeruginosa Enhances Killing of Phagocytes by Up-Regulating the Type III Secretion System in Ciprofloxacin Induced Persister Cells

Mei Li(Jiangsu University), Weihui Wu(Nankai University)
Frontiers in Cellular and Infection Microbiology
October 14, 2016
10.3389/fcimb.2016.00125
Cited by 60

Related Papers

TMEM43/LUMA is a key signaling component mediating EGFR-induced NF-κB activation and tumor progression
|Oncogene|2016|52
Regulatory Role of PopN and Its Interacting Partners in Type III Secretion of<i>Pseudomonas aeruginosa</i>
|Journal of Bacteriology|2007|50
Two novel mutations in TMEM38B result in rare autosomal recessive osteogenesis imperfecta
|Journal of Human Genetics|2016|43
Novel Mutations in FKBP10 and PLOD2 Cause Rare Bruck Syndrome in Chinese Patients
|PLoS ONE|2014|35
Novel Mutations in PLOD2 Cause Rare Bruck Syndrome
|Calcified Tissue International|2017|26