Chemoresistance in Pancreatic Cancer Is Driven by Stroma-Derived Insulin-Like Growth Factors
Lucy Ireland(University of Liverpool), Ainhoa Mielgo(University of Liverpool), Sarah E. Coupland(University of Liverpool), Carolyn Rainer(University of Liverpool), Ulrike Weyer-Czernilofsky(Boehringer Ingelheim (Austria)), Sebastian R. Nielsen(Icahn School of Medicine at Mount Sinai), Pedro A. Pérez–Mancera(University of Liverpool), Michael C. Schmid(University of Liverpool), Azzam Taktak(University of Liverpool), Muhammad S. Ahmed(University of Liverpool), Dannielle D. Engle(Cold Spring Harbor Laboratory), David A. Tuveson(Cold Spring Harbor Laboratory), Fiona Campbell(University of Birmingham), Valeria Quaranta(University of Liverpool), Almudena Santos(University of Liverpool), Thomas Bogenrieder(Boehringer Ingelheim (Austria))
Cited by 249
Related Papers
Distinct populations of inflammatory fibroblasts and myofibroblasts in pancreatic cancer
|The Journal of Experimental Medicine|2017|2.4k
What is meant by intention to treat analysis? Survey of published randomised controlled trials
|BMJ|1999|2.2k
Organoid Models of Human and Mouse Ductal Pancreatic Cancer
|Cell|2014|2.2k
Analysis of lung tumor initiation and progression using conditional expression of oncogenic <i>K-ras</i>
|Genes & Development|2001|2k
Restoration of p53 function leads to tumour regression in vivo
|Nature|2007|1.8k