Investigating the potential role of non-vls genes on linear plasmid 28–1 in virulence and persistence by Borrelia burgdorferiPetronella R. Hove Magunda(Washington State University), Troy Bankhead(Washington State University)BMC MicrobiologyAugust 8, 201610.1186/s12866-016-0806-4Cited by 14SaveCiteExport RISWatch citationsRelated PapersBreaking a barrier: In trans vlsE recombination and genetic manipulation of the native vlsE gene of the Lyme disease pathogen|PLoS Pathogens|2025|3