Long‐term administration of the mitochondria‐targeted antioxidant mitoquinone mesylate fails to attenuate age‐related oxidative damage or rescue the loss of muscle mass and function associated with aging of skeletal muscle
Giorgos K. Sakellariou(Culham Science Centre), Malcolm J. Jackson(University of Liverpool), Gareth Nye(Arthritis UK), Anne McArdle(University of Liverpool), Richard Griffiths(University of Liverpool), Ifigeneia Giakoumaki(Arthritis UK), Nicola Wells(Arthritis UK), Adam P. Lightfoot(Manchester Metropolitan University), Tim Pearson(Taronga Conservation Society Australia)
Cited by 51
Related Papers
Metabolic and nutritional support of critically ill patients: consensus and controversies
|Critical Care|2015|412
Studies of Mitochondrial and Nonmitochondrial Sources Implicate Nicotinamide Adenine Dinucleotide Phosphate Oxidase(s) in the Increased Skeletal Muscle Superoxide Generation That Occurs During Contractile Activity
|Antioxidants and Redox Signaling|2012|281
Redefining the major contributors to superoxide production in contracting skeletal muscle. The role of NAD(P)H oxidases
|Free Radical Research|2013|181
Time course and differential responses of the major heat shock protein families in human skeletal muscle following acute nondamaging treadmill exercise
|Journal of Applied Physiology|2006|165
Mitochondrial ROS regulate oxidative damage and mitophagy but not age-related muscle fiber atrophy
|Scientific Reports|2016|135