Baicalin suppresses NLRP3 inflammasome and nuclear factor-kappa B (NF-κB) signaling during Haemophilus parasuis infection
Shulin Fu(Wuhan Polytechnic University), Chien‐An Andy Hu(University of New Mexico)
Cited by 65
Related Papers
Apolipoprotein L1, a Novel Bcl-2 Homology Domain 3-only Lipid-binding Protein, Induces Autophagic Cell Death
|Journal of Biological Chemistry|2008|218
PUMA Overexpression Induces Reactive Oxygen Species Generation and Proteasome-Mediated Stathmin Degradation in Colorectal Cancer Cells
|Cancer Research|2005|121
Apolipoprotein L6, a Novel Proapoptotic Bcl-2 Homology 3–Only Protein, Induces Mitochondria-Mediated Apoptosis in Cancer Cells
|Molecular Cancer Research|2005|109
Baicalin modulates NF-κB and NLRP3 inflammasome signaling in porcine aortic vascular endothelial cells Infected by Haemophilus parasuis Causing Glässer’s disease
|Scientific Reports|2018|44
The effects of baicalin on piglets challenged with Glaesserella parasuis
|Veterinary Research|2020|42