Alternative Ways to Die5Epac1 deletion prevents cardiomyocyte apoptosis during ischemia/reperfusion6Subcellular redistribution of mitogen and stress activated kinase 1 (MSK1) contributes to protection against oxidative stress- induced apoptosis in cardiac myocytes7Excessive ROS production in mitochondria switches off protective mitochondrial kinase signaling
Marion Laudette(University of Gothenburg), Tetsuji Miura(Sapporo Medical University), Frank Lezoualc’h(Inserm), Kyriakos Mellidis(Aristotle University of Thessaloniki), Masaya Tanno(Sapporo Medical University), Apostolopoulos P Karameris A(Aristotle University of Thessaloniki), Pierre Sicard(Centre National de la Recherche Scientifique), Florence Tortosa(Inserm), Masashi Mizuno(Sapporo Medical University), Yuki Tatekoshi(Sapporo Medical University), Jeanne Mialet‐Perez(Centre National de la Recherche Scientifique), A Moraiti(Aristotle University of Thessaloniki), Loubina Fazal(Inserm), Atsushi Kuno(Okayama University of Science), Satoko Ishikawa(Kanazawa University Hospital), Sandrine Pons(École Nationale Vétérinaire d'Alfort), Antigone Lazou(Aristotle University of Thessaloniki), Keitaro Nishizawa(Sapporo Medical University), Wataru Ohwada(Sapporo Medical University), Toshiyuki Yano(Sapporo Medical University), Bijan Ghaleh(Inserm), Takayuki Miki, Eleftheria Barlaka(Aristotle University of Thessaloniki)
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