Positive and negative early life experiences differentially modulate long term survival and amyloid protein levels in a mouse model of Alzheimer's disease
Sylvie L. Lesuis(Netherlands Institute for Neuroscience), Harm J. Krugers(University of Amsterdam), Paul J. Lucassen(Netherlands Institute for Neuroscience), Hervé Maurin(Institute of Pathology and Genetics), Peter Borghgraef(Allen Institute for Brain Science), Fred Van Leuven(KU Leuven)
Cited by 61
Related Papers
Early Phenotypic Changes in Transgenic Mice That Overexpress Different Mutants of Amyloid Precursor Protein in Brain
|Journal of Biological Chemistry|1999|668
Maternal Care and Hippocampal Plasticity: Evidence for Experience-Dependent Structural Plasticity, Altered Synaptic Functioning, and Differential Responsiveness to Glucocorticoids and Stress
|Journal of Neuroscience|2008|658
A disintegrin-metalloproteinase prevents amyloid plaque formation and hippocampal defects in an Alzheimer disease mouse model
|Journal of Clinical Investigation|2004|622
Presenilin 2 deficiency causes a mild pulmonary phenotype and no changes in amyloid precursor protein processing but enhances the embryonic lethal phenotype of presenilin 1 deficiency
|Proceedings of the National Academy of Sciences|1999|523