Human glia can both induce and rescue aspects of disease phenotype in Huntington disease
Abdellatif Benraiss(University of Rochester Medical Center), Steven A. Goldman(University of Copenhagen)
Cited by 201
Related Papers
A TCF7L2-responsive suppression of both homeostatic and compensatory remyelination in Huntington disease mice
|Cell Reports|2022|29
Charting the transition from in vitro gliogenesis to the in vivo maturation of transplanted human glial progenitor cells
|bioRxiv (Cold Spring Harbor Laboratory)|2025|1
In vivo selection and glymphatic delivery of AAV5 capsids engineered to target human glial progenitor cells
|bioRxiv (Cold Spring Harbor Laboratory)|2025|1